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Ziconotide

Ziconotide is a synthetic omega-conotoxin MVIIA peptide that blocks N-type calcium channels, approved for severe chronic pain via intrathecal delivery.

By Encyclopeptide Editorial | 2 min read
conotoxin calcium-channel pain intrathecal omega-conotoxin

Chemical Identity

PropertyValue
Chemical FormulaC102H172N36O32S7
Molecular Weight2639.14 g/mol
CAS Number107452-89-1
IUPAC NameSynthetic omega-conopeptide MVIIA
Peptide ClassN-type Calcium Channel Blocker
Sequence HomologySynthetic version of Conus magus toxin

Structure

Ziconotide is a synthetic 25-amino acid peptide identical to omega-conotoxin MVIIA from the venom of the marine cone snail Conus magus. It contains three intramolecular disulfide bonds forming a compact, rigid structure. The peptide belongs to the omega-conotoxin family characterized by a cysteine knot motif.

Mechanism of Action

Ziconotide selectively blocks N-type voltage-gated calcium channels (Cav2.2) on primary afferent neurons in the dorsal horn of the spinal cord. By inhibiting calcium influx at presynaptic terminals, it prevents the release of pro-nociceptive neurotransmitters including substance P, glutamate, and CGRP. Unlike opioid analgesics, ziconotide does not produce tolerance or physical dependence.

Clinical Applications

Ziconotide (Prialt) is approved for:

  • Severe chronic pain: Management of severe chronic pain in patients who require intrathecal therapy
  • Cancer pain: Refractory cancer pain unresponsive to other treatments
  • Non-malignant chronic pain: Failed back surgery syndrome, complex regional pain syndrome

Administered exclusively via continuous intrathecal infusion using an implanted pump, as systemic administration causes intolerable side effects.

Pharmacokinetics

  • Half-life: 2.9-6.5 hours (intrathecal)
  • Tmax: Immediate (intrathecal)
  • Bioavailability: 100% intrathecally; negligible systemic bioavailability
  • Metabolism: Proteolytic degradation in CSF and systemic circulation
  • Distribution: Confined to CSF after intrathecal administration

Safety and Side Effects

Adverse effects include dizziness (30%), nausea (27%), confusion (11%), abnormal gait (9%), memory impairment (6%), and psychiatric symptoms (hallucinations, paranoia). Contraindicated in patients with pre-existing psychosis or those receiving intrathecal infusions via non-proprietary pumps. Must be titrated slowly to minimize CNS adverse effects. Not an opioid; no abuse potential.

References

  • Staats, P.S., et al. (2004). Intrathecal ziconotide in the treatment of refractory pain in patients with cancer or AIDS. JAMA, 291, 63-70.
  • Miljanich, G.P. (2004). Ziconotide: neuronal calcium channel blocker for treating severe chronic pain. Current Medicinal Chemistry, 11, 3029-3040.

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